Systemic laupus case study case study

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Paper type: Health,

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Anemia, Examine Guide, Circumstance Studies, Case Study

Excerpt from Case Study:

Systemic Laupus Erythematosus Example

Systemic Lupus Erythematosus

Systemic lupus erythematosus (SLE) is known as a chronic, life-long autoimmune disease that affects multiple tissues and organs in the body (Madhok and Wu, 2009). The primary tissues and bodily organs affected by immune system dysfunction are the joints, kidneys, vessel wall space, and skin. SLE is definitely believed to be due to an connection between genetic factors and environmental sets off. The disease is likewise more prevalent among women, which is like possibility that hormones play some role in disease etiology.

Of primary matter is laupus glomerulonephritis (Madhok and Wu, 2009). Other serious problems include heart and neuropsychiatric involvement. Many patients can experience osteoarthritis or joint pain (84%), fever (52%), malar allergy (58%), and photosensitivity (45%). Accordingly, a diagnosis of SLE will depend in part on a blood vessels test pertaining to anti-nuclear antibodies (ANA) and presentation with rash, joint pain, tiredness, and/or low-grade fevers.

The pain in Vicki’s knees, shoulders, and elbows may be the result of inflammation, which is due to an autoimmune reaction to cartilaginous tissue inside the joints (A. D. A. M., 2013). The fever is most likely low grade and due to systemic inflammation plus the frequent infections is the response to immune dysfunction. Inflammation is likewise the cause of the prototypical butterfly rash. The anemia may be the result of flat iron deficiency, possibly due to regular menstruation or perhaps gastrointestinal blood loss; however hemolytic anemia is yet another possibility. Hemolytic anemia has been associated with quite high serum numbers of cardiolipin auto-antibodies. The hardening of the skin area over the fingertips is not really a symptom of SLE, but of scleroderma, an additional autoimmune disease. Scleroderma is due to excessive collagen deposition reminiscent of what happens during wound restore. SLE and scleroderma may co-occur.

Laboratory tests probably will reveal the next:

1 . CHOIX titer previously mentioned 1: forty five (Gill, Quisell, Rocca, and Walters, 2003)

2 . CBC revealing less than normal RBC count, hemoglobin, and hematocrit indicating straightener deficiency (Schrier, 2013) or possibly a positive erythrocyte antibody display screen consistent with a diagnosis of hemolytic anemia (ABSCM) (Mayo Medical Laboratories, 2013)

3. CBC with differential box showing a low white blood cell depend

Renal inflammation due to intensifying SLE could be devastating. Near to 50% of SLE sufferers will experience nephritis as a result of deposition of

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